Salt stress is one of the main factors which currently reduce plant productivity.Salinity produce osmotic stress by decreasing the chemical activity of water and a specific problem is ion toxicity, because high intracelular concentations of chloride and Na⁺ are deleterious to cellular system. Indeed osmotic stress induces oxidative stress and to control the level of ROS and to protect cells under stress conditions, plant tissues contain several enzymes scavenging ROS (Superoxide dismutases, catalases, guaiacol type peroxidases and ascorbate peroxidases). The aim of this work was to investigate the effect of salt stress upon membrane lipid peroxidation, ascorbate peroxidase (APX) and guaicol peroxidase (GPX) activities on roots and leaves from Vigna unguiculata (L.) Walp cv. Vita 3. V. unguiculata seeds were dark germinated, at 28°C, during 3 days and than transferred to a growth chamber with a photoperiodism of 12 hours in Hoagland's medium and grown in the absence (control) or presence of NaCl (100 mM) for two weeks. The degree of lipid peroxidation was the measure of malondialdehide production by Calmak & Horst (1991) as an oxidative stress marker. The APX medium contained 36 mM phosphate buffer pH 6.0, 36 mM EDTA, 1mM H₂O₂, 0.5mM ascorbate and 3mg/mL of plant extract and the GPX medium contained 63 mM phosphate buffer pH 7.0, 63 mM EDTA, 5 mM Guaiacol, 15 mM H₂O₂ and 0,5mg/mL of the plat extract. The APX and GPX activities were measured spectrophotometrically at 290nm and 470nm respectively. Salinity produced oxidative stress, indicated by malondialdehide level that was detected only in salt stress condition. In salt-treated plants, the APX and GPX activities decreased 11 % and 33 % in roots and 11% and 1283 % in leaves respectively. These results reveal that roots and leaves from V. unguiculata under salt stress were not protected by APX and GPX against oxidative damage, suggesting that another ROS scavenging system should be induced.